Raisner, R.M., Hartley, P.D., Meneghini, M.D., Bao, M.Z., Liu, C.L., Schreiber, S.L., Rando, O.J., and Madhani, H.D. (2005).

Histone variant H2A.Z marks the 5' end of both active and inactive genes in euchromatin.

Cell. 123(2): 233-248.


Kobor, M.S., Venkatasubrahmanyam, S., Meneghini, M.D., Gin, J.G., Jennings, J.L., Link, A.J., Madhani, H.D., and Rine, J. (2004).

A Protein Complex Containing the Conserved Swi2/Snf2-Related ATPase Swr1p Deposits Histone Variant H2A.Z into Euchromatin.

PLoS Biol. 2(5): E131.


Meneghini, M.D., Wu, M., and Madhani , H. D. (2003).

Conserved histone variant H2A.Z protects euchromatin from the ectopic spread of silent heterochromatin.

Cell. 112(5): 725-736.


Maduro, M.F., Meneghini, M.D., Bowerman, B., Broitman-Maduro, G., and Rothman, J.H. (2001).

Restriction of mesendoderm to a single blastomere by the combined action of SKN-1 and a GSK-3ß homolog is mediated by MED-1 and -2 in C. elegans.

Molecular Cell. 7(3): 475-485.

Schlesinger, A., Shelton, C.A., Maloof, J.N., Meneghini, M., and Bowerman, B. (2000).

Wnt pathway components orient a mitotic spindle in the early Caenorhabditis elegans embryo without requiring transcription in the responding cell.

Genes & Dev. 13: 2028-2038.


Meneghini, M.D., Ishitani, T., Carter, J.C., Hisamoto, N., Ninomiya-Tsuji, J., Thorpe, C.J., Hamill, D.R., Matsumoto, K., and Bowerman, B. (1999).

MAP kinase and Wnt pathways converge to downregulate an HMG-domain repressor in Caenorhabditis elegans.

Nature, 399:793-797.


Ishitani, T., Ninomiya-Tsuji, J., Nagai, S., Nishita, M., Meneghini, M., Barker, N., Waterman, M., Bowerman, B., Clevers, H., Shibuya, H., and Matsumoto, K. (1999).

The TAK1-NLK-MAPK-related pathway antagonizes signalling between ß-catenin and transcription factor TCF.

Nature, 399: 798-802


Keesee, S.K., Meneghini, M.D., Szaro, R.P., and Wu, Y.J. (1994).

Nuclear matrix proteins in human colon cancer.

Proc. Natl. Acad. Sci. U.S.A. 91: 1913-1916.


Miller, T., Beausang, L.A., Meneghini, M., and Lidgard, G. (1993).

Death-induced changes to the nuclear matrix: the use of anti-nuclear matrix antibodies to study agents of apoptosis.

Biotechniques, 15: 1042-1047.